1 debridements (range, 1-4 debridements).
Conclusions. The presence of a ventriculoperitoneal shunt is a statistically significant risk factor for wound infection after corrective surgery for neuromuscular scoliosis. Wound infection is associated with pseudarthrosis and prolonged hospitalization.”
“Ionic liquids (ILs) functionalized GSK1120212 inhibitor solvent impregnated resins (SIRs)
were prepared using IL modified Merrified resin as the polymeric supports by impregnation of extractant for extraction of Sc(III). The ILs modified Merrifield resin were prepared via covalent anchoring of imidazolium salts onto Merrifield resin. The polymeric supports with imidazolium chloride group (RCl) and imidazolium hexafluorophosphate group (RPF6) were characterized by FTIR, TGA, and elemental analysis. It was found that RCl and RPF6 had tunable hydrophilicity and hydrophobicity, different acid stability, and swelling behaviors in solvents. The effect of Cyanex 923 extractant or [C(8)mim][PF6] IL impregnated on RCl and RPF6 were studied. selleck chemicals The results showed Cyanex 923 and [C(8)mim][PF6] exhibited stronger affinity to RPF6 than to RCl. RPF6 with Cyanex 923 was found to be effective in Sc(III) extraction. The extraction mechanisms of SIRs containing RPF6 and Cyanex 923 with or without [C(8)mim][PF6] were cation exchange
and neutral complexation, respectively. MEK inhibitor side effects [C(8)mim][PF6] acted as an extraction media and was involved in the cation exchanged extraction reaction. Sc(III) can be selectively separated from Tm(III), Yb(III), and
Lu(III) by the SIRs. (C) 2011 Wiley Periodicals, Inc. J Appl Polym Sci 120: 3284-3290, 2011″
“The Philadelphia (Ph) chromosome and/or Breakpoint cluster region-Abelson leukemia virus oncogene transcript are unique markers for chronic myeloid leukemia (CML). However, CML demonstrates heterogeneous presentations and outcomes. We analyzed the cytogenetic and molecular results of CML patients to evaluate their correlation with clinical presentations and outcome. A total of 84 newly diagnosed CML patients were enrolled in the study. Patients were treated according to disease status. Bone marrow samples were obtained to perform cytogenetic and molecular studies. Clinical presentations, treatment courses, and survival were reviewed retrospectively. Among 84 patients, 72 had chronic phase and 12 had accelerated phase CML. Cytogenetic study showed 69 (82.1%) with the classic Ph chromosome, 6 (7.2%) with a variant Ph chromosome, and 9 (10.7%) with additional chromosome abnormalities. Fifty-four (64.3%) cases harbored b3a2 transcripts, 29 (34.5%) had b2a2 transcript, and 1 had e19a2 transcript. There was no difference in clinical presentations between different cytogenetic and molecular groups; however, additional chromosome abnormalities were significantly associated with the accelerated phase.