It should be taken into account, too, that the light path in Wnt inhibition typical measuring chambers (usually 1–2 cm) is much smaller than that in the culture vessel (5–10 cm), so that the light intensity reaching every single cell is selleck higher due to less self-shading of the cells. The use of O2 electrodes of the Clark type is a common technology which will not be

explained here in detail. It should be noted, however, that Clark-type electrodes can easily be converted to H2 electrodes just by applying a different potential. Details of assembling and using these electrodes are given in references Wang (1980), Kuroda et al. (1991), and Takeshita et al. (1993). An easy method to analyze in vivo H2-production rates of illuminated C. reinhardtii cells without a H2 electrode will

be described here. This technique is suitable to determine the real H2-evolution rate of the cells, which can be only roughly concluded from the accumulation of H2 in the gas phase of the incubation LCZ696 flask or in a gas trap (see below). For this purpose, a 2-ml sample of the main culture is taken with a syringe by piercing through the septum and gently injected through the rubber seal of an 8–10 ml headspace bottle as described above, which has been gassed with Ar before. The little vessels are then placed in the light. The cell suspension has to be rocked or stirred so that the cells do not settle. A shaking water bath made from plexiglas standing on top a light source is optimal. After 10 min, a volume of the gas phase is analyzed with a gas chromatograph to determine H2 concentration. Then, the cells are incubated for 1 h, and H2 is detected again. The difference of the H2 concentration in the beginning and after 60 min is the amount of H2 that has been produced by the cells. It should be noted that the 10 min of pre-incubation is applied to let the cells adapt to the system, which will differ from the incubation conditions of the main culture in some aspects. Furthermore, during the

transfer of the cells, some air (i.e., O2) might have entered the cell suspension, and the cells might have been shaded to some extent. Non-specific serine/threonine protein kinase During the pre-incubation, the algae will stabilize their H2 metabolism. The first analysis of the H2 concentration after the 10 min duration is important to take into account the H2 which has been produced during this pre-incubation phase and the gas which was introduced into the reaction vessel by the algal suspension. In the active H2-producing phase of S-deprived C. reinhardtii cultures, significant amounts of H2 are dissolved in the medium of the cells. The above point should also be kept in mind when carrying out in vitro hydrogenase activity assays with S-depleted algae.

Free serosanguineous fluid as a result of haemorrhagic extravasio

Free serosanguineous fluid as a result of haemorrhagic extravasion is a characteristic finding in the peritoneal cavity. In the literature the treatment of choice included additional appendicectomy to prevent future diagnostic problems. Successful conservative management has also been reported [5, 13]. Histology findings of haemorrhagic infarction and fat necrosis confirm

the diagnosis with the presence of fibrosis indicative of a longer disease process [4]. The prognosis for primary omental torsion is good with fast post operative recovery and minimal morbidity. The natural disease progress if left untreated will result in fibrosis, necrosis and occasional autoamputation and clinical improvement [7, 14]. Selleck Brigatinib Prognosis in secondary torsion depends in the underlying pathology. Left sided omental torsion may be commonly misdiagnosed as diverticulutis and managed conservatively, resulting in less common diagnosis [7]. Conclusion Omental torsion presents with non specific symptoms of an acute abdomen and is mainly diagnosed intraoperatively during diagnostic laparoscopy. Awareness of omental torsion as a differential diagnosis in the acute abdomen and careful inspection

of omentum in a “”negative laparoscopy”" are Doramapimod supplier recommended for appropriate management of the surgical patient [4]. However cases without complications, may be managed conservatively in future [10]. Consent Written informed consent was obtained from the patient for publication of this case report. References 1. Theriot JA, Sayat J, Franco S, Buchino JJ: Childhood obesity: a risk factor for omental torsion. Pediatrics 2003,112(6 Pt 1):e460.CrossRefPubMed 2. Saber A, LaRaja R: Omental Torsion. [http://​emedicine.​medscape.​com] EMedicine, article 191817 2007. 3. Eitel GG: Rare omental torsion. NY Med Rec 55 1899, Rebamipide 715. 4. Parr NJ, Crosbie RB: Intermittent omental torsion–an unusual cause of recurrent GSK690693 clinical trial abdominal pain? Postgraduate Medical Journal 1989, 65:114–115.CrossRefPubMed 5. Tsironis A, Zikos N, Bali C, Pappas-Gogos G, Koulas S, Katsamakis N: Primary Torsion of the Greater Omentum: Report of Two

Cases and Review of the Literature. The Internet Journal of Surgery 2008.,17(2): 6. Al-Jaberi T, Gharaibeh K, Yaghan R: Torsion of abdominal appendages presenting with acute abdominal pain. Annals of Saudi Medicine 2000.,20(3–4): 7. Jeganathan R, Epanomeritakis E, Diamond T: Primary torsion of the omentum. Ulster Med J 2002,71(1):76–7.PubMed 8. Atar E, Herskovitz P, Powsner E, Katz M: Primary greater omental torsion: CT diagnosis in an elderly woman. Isr Med Assoc J 2004,6(1):57–8.PubMed 9. Parr NJ, Crosbie RB: Intermittent omental torsion–an unusual cause of recurrent abdominal pain? Postgrad Med J 1989,65(760):114–5.CrossRefPubMed 10. Abdennasser el K, Driss B, Abdellatif D, Mehci A, Souad C, Mohamed B: Omental torsion and infarction: CT appearance. Intern Med 2008,47(1):73–4.CrossRefPubMed 11.

CA15-3 monoclonal antibody was purchased from Zhongshan Goldenbri

CA15-3 monoclonal antibody was purchased from Zhongshan Goldenbridge Biotechnology (Beijing, China). A Facsvantage SE flow cytometer was purchased from BD Company (USA); the Gel Doc XR quantity one gel image analyzer was purchased from Bio-Rad Company (Hercules, CA, USA) and the CX40 fluorescent invert microscope was purchased from Olympus (Tokyo, Japan). 1.3 Cell culture and

nude mice breeding A Histone Methyltransferase inhibitor female breast cancer patient, aged 72 years, without chemotherapy and particular previous medical history, was treated by Breast & Thyroid & Pancreas Surgery selleck chemical in Second Affiliated Hospital of Chongqing Medical University. A specimen was taken from the patient’s breast, which had undergone radical mastectomy. The pathology results revealed an infiltrating ductal carcinoma; immunohistochemistry revealed ER (+), PR(++), CerbB-2(-). The breast carcinoma specimen was sent to the lab within

2 h and cut into 1-mm3 pieces. The sample was digested for 12 h in a mixture of 1% collagenase II plus hyaluronidase at 37°C, the supernatant was discarded, and the sample without supernatant was centrifuged at 1000 r/min for 5 min. A single breast carcinoma cells was collected, diluted to a concentration of 105/mL, and then cultured in RPMI 1640 + 10% fetal bovine serum culture medium. Trypan blue stain was used to assess cell viability, and vivid breast carcinoma cells were taken to descendence. Cell adherence was used repeatedly to remove cell impurities [6].

Human breast cancer cell line MDA-MB-231 was cultured in RPMI-1640 medium plus 10% fetal bovine XL184 serum, 100 U/mL penicillin, and 100 mg/L streptomycin at 37°C in an incubator with 5% CO2 and saturated in a humidity environment. The cultured cells within logarithmic growth were used in this study. Cell suspensions were prepared Sulfite dehydrogenase by trypsin digestion. Nude mice were kept in a specific pathogen free environment with a temperature of 22-25°C and 50-65% humidity. Drinking water, feed, and experimental materials were disinfected by sterilization, and the rule of aseptic operation was strictly followed. Our research reported in the manuscript has been performed with the approval of Chongqing Medical University ethics committee. 1.4 Immunocytochemical fluorescent staining For fluorescent staining, 1 × 105 cultured cells were planted onto cover glass. The cover glass was removed when the cells covered 80% of the glass. After being fixed, the cover glass was 1) used to hatch inactive endogenous enzyme, 2) treated in 0.1% Triton liquid, 3) washed within phosphate-buffered saline (PBS), 4) subjected to immunocytochemical and immunofluorescent staining according to instructions for CA15-3 primary antibody (1:100) and fluorescein isothiocyanate-marked secondary antibody (1:100), 5) sealed with glycerine, 6) inserted into an Olympus CX40 inverted microscope for observation and recording. 1.5 Grouping and drug administration 1.5.

8227 0 0127 0 9091 AUC0–inf 0 8255 0 0099 0 9010 C max 0 5835 0 1

8227 0.0127 0.9091 AUC0–inf 0.8255 0.0099 0.9010 C max 0.5835 0.1291 0.8606 AUC 0–inf area under the serum concentration–time curve from time zero to infinity AUC 0–t area under the serum concentration–time curve from time zero to time of last measurable concentration, C max maximum serum concentration Fig. 2 Mean plasma ibandronic acid concentrations obtained for the test and reference formulations following a 150-mg dose (log scale). N = 146 for ibandronic acid, N = 146 for

Bonviva® (first administration), N = 142 for Bonviva® (second administration), EDTA Ethylene diaminetetraacetic acid The CVWR for AUC0–t , AUC0–inf and C max were 39.77, 39.45 and 43.23 %, respectively. The limits of the acceptance range buy INK1197 based upon the within-subject variability seen in the bioequivalence study using scaled average bioequivalence were 73.01–136.97 %. No statistical outliers were detected for the reference formulation following examination A-1155463 concentration of the distribution of the ln-transformed C max. The 90 % confidence selleck chemicals intervals were 95.05–110.67 for

C max, 94.35–107.94 for AUC0–t and 94.37–107.88 for AUC0–inf, which are within the predefined bioequivalence acceptance range of 80.00–125.00 %. For C max, the observed ratio and confidence intervals were also within the limits of acceptance obtained using the scaled average bioequivalence Farnesyltransferase approach. Wilcoxon’s test performed on the

t max data showed no statistically significant difference between treatments (p = 0.1382). The least-squares means ratios, the 90 % geometric confidence intervals, and the CVWR for the reference product are presented in Table 4. Table 4 Ibandronic acid: ratios, 90 % geometric confidence intervals (CI) for AUC0–t , AUC0–inf and C max and intra-subject CV for Bonviva® Variable Treatment comparisons Ratioa (%) 90 % CIb (%) Intra-subject CV (Bonviva®) (%) AUC0–t Test (A)—reference (B) 100.92 94.35–107.94 39.77 AUC0–inf Test (A)—reference (B) 100.90 94.37–107.88 39.45 C max c Test (A)—reference (B) 102.56 95.05–110.67 43.23 aCalculated using least-squares means b90 % geometric confidence interval using ln-transformed data cThe scaled average bioequivalence approach was used for C max and the widened limits obtained were 73.01–136.97 % AUC 0–inf area under the serum concentration–time curve from time zero to infinity AUC 0–t area under the serum concentration–time curve from time zero to time of last measurable concentration, C max maximum serum concentration, CV coefficient of variance 3.

570 m, on cut log of Picea abies 120 cm thick, 2 5 m above ground

570 m, on cut log of Picea abies 120 cm thick, 2.5 m above ground, in a pile stored at roadside, soc. Trichaptum abietinum, 8 Oct. 2004, W. Jaklitsch, W.J. 2774 (WU 24017; isolate C.P.K. 2001). Czech Republic, South Bohemia, at roadside 5.7 km north from Frymburk, MTB 7250/4, 48°42′36″ N, 14°08′06″ E, elev. 750 m, on partly decorticated cut log of Picea abies 22 cm thick, on the ground, protected by grass, herbs, soc. Neonectria fuckeliana, Stereum sanguinolentum, Sarea

resinae, immature, culture from conidia, 22 Sep. 2003, W. Jaklitsch, W.J. 2408 (WU 24010; culture C.P.K. 965); 2.7 km before Frymburk approaching from Lipno, MTB 7351/3, 48°38′22″ N, 14°10′52″ E, elev. 740 m, on partly decorticated logs of Pinus sylvestris 10–43 cm thick, stored in a pile at the roadside, mostly immature, 3 Oct. 2004, W. Jaklitsch, W.J. 2758 (WU 24014; culture C.P.K. Sepantronium clinical trial 1998). France, La Moselle, Parc Lorraine, Héming, between Étang du Stock and Maizières de Vic, ICG-001 price 48°43′35″ N, 06°54′07″ E, elev. 180 m, on cut and mostly corticated branches and logs of Quercus robur 2–40 cm thick, on bare ground or squeezed into moist soil, soc. Amphiporthe leiphaemia, Diatrypella sp., Bulgaria inquinans, part attacked by white mould, 5 Sep. 2004, W. this website Jaklitsch & H. Voglmayr, W.J. 2677 (WU 24011; culture C.P.K. 1995). Germany, Bavaria, Unterfranken, Landkreis Haßberge, Haßfurt, close to Mariaburghausen, left roadside heading from Knetzgau to Haßfurt, MTB 5929/3, 50°00′31″

N, 10°31′17″ E, elev. 270 m, on cut branches of Quercus robur 4–5 below cm thick, on bark, holomorph, teleomorph immature, 29 Aug. 2006, W. Jaklitsch & H. Voglmayr,

W.J. 2962 (WU 29457, culture C.P.K. 2457). Nordrhein-Westfalen, Märkischer Kreis, Plettenberg-Böddinghausen, Naturschutzgebiet Bommecketal, 1 km south from the entrance to the nature reserve, MTB 4713/3, elev. 300 m, on corticated log of Fraxinus excelsior 15 cm thick, on bark, soc. Neonectria coccinea, 8 Oct. 2006, K. Siepe & F. Kasparek, W.J. 3061 (WU 29459, culture C.P.K. 2867). Netherlands, Putten, in Armen Bos of the arboretum Landgoed Schovenhorst, elev. 0 m, on corticated branch of Quercus robur 4–10 cm thick, on bark, holomorph, teleomorph immature, 19 Nov. 2006, H. Voglmayr, W.J. 3048 (WU 29458, culture C.P.K. 2856). Sweden, Uppsala Län, Fredrikslund, pine forest near nature reserve Kungshamn-Morga, 1.5 km NE of Fredrikslund, 59°47′00″ N, 17°39′00″ E, elev. 50 m, on cut and mostly corticated tree tops and branches of Pinus sylvestris 5–9 cm thick, on the ground, 8 Oct. 2003, W. Jaklitsch & S. Ryman, W.J. 2450 (BPI 872089; cultures CBS 119326 = C.P.K. 984, G.J.S. 04-21 from white stroma). United Kingdom, Derbyshire, Baslow, Longshaw Country Park, Peak District National Park, 53°18′26″ N, 01°36′08″W, elev. 350 m, on corticated branches and logs of Acer pseudoplatanus 2–10 cm thick, on the ground in open grassland, holomorph, teleomorph immature, culture from conidia, 10 Sep.

Recently, a selC-associated genomic island of APEC strain BEN2908

Recently, a selC-associated genomic island of APEC strain BEN2908 was found to be involved in carbohydrate uptake and virulence [8]. Also in the same APEC strain, a carbohydrate metabolic operon (frz) that is highly associated with ExPEC promotes fitness under stressful conditions and invasion of eukaryotic cells [33]. Our STM results showed that one tkt1 STM-mutant was out-competed by the wild type from two to a thousand fold in lung, heart, liver, kidney and spleen

of 5-week-old chickens. The functional analysis using phenotype microarray revealed that a tkt1 mutant has defects in use of Pro-Ala or Phe-Ala as a nitrogen source. These results selleck kinase inhibitor strongly suggest that tkt1 is involved in bipeptide metabolism and contributes to fitness and virulence of APEC. Interestingly, dipeptide transport proteins, DppA and OppA, were identified to be selleck chemicals llc up-regulated when UPEC strain CFT073 was cultured in human urine compared to CFT073 cultured in LB depleted with iron [34]. The greatest challenges confronted by bacterial pathogens are environmental changes, including the rapid changes they encounter in nutrient availability [35]. In the course of evolution, pathogenic organisms have developed several mechanisms to sense and utilize available nutrient

sources associated with particular niches or to favor the most efficiently metabolizable AZD9291 in vivo nutrient sources when exposed to a range of choices [36]. Thus, genes involved in metabolism, which are required for bacterial growth in specific infection sites, contribute to fitness and virulence. On the other hand, the efficiency of metabolism of a nutrient source or the presence of a specific nutrient source might serve as a signal to switch ‘on’ or ‘off’ specific virulence genes in particular infection niches [36]. Conclusions A previously identified virulence-associated gene tkt1 was further characterized

in this CYTH4 study. The results demonstrated that this gene is strongly associated with ExPEC strains of phylogenetic group B2 from human and avian origin and is localized in a genomic island. Function analyses showed that Tkt1 has very little transketolase activity and seems to be involved in peptide nitrogen metabolism. Acknowledgements This work was supported by USDA NRICGP Microbial Functional Genomics Program (20083560418805). References 1. Russo TA, Johnson JR: Proposal for a new inclusive designation for extraintestinal pathogenic isolates of Escherichia coli : ExPEC. J Infect Dis 2000,181(5):1753–1754.PubMedCrossRef 2. Dziva F, Stevens MP: Colibacillosis in poultry: unravelling the molecular basis of virulence of avian pathogenic Escherichia coli in their natural hosts. Avian Pathol 2008,37(4):355–366.PubMedCrossRef 3. Li G, Tivendale KA, Liu P, Feng Y, Wannemuehler Y, Cai W, Mangiamele P, Johnson TJ, Constantinidou C, Penn CW, et al.: Transcriptome analysis of avian pathogenic Escherichia coli O1 in chicken serum reveals adaptive responses to systemic infection.

So, the establishment of an excess minority carrier hole in

So, the establishment of an excess minority carrier hole in

the vicinity is observed [28]. The current moves mainly from the drain to the source which consists of both drift and diffusion currents. The created 2D anticipated framework is expected to cause an explicit analytical current equation in the subthreshold system. Considering the weak Captisol research buy inversion region, the diffusion current is mainly dominated and relative to the electron absorption at the virtual cathode [47]. A GNR FET is a voltage-controlled tunnel barrier device for both the Schottky and doped contacts. The drain current through the barrier consists of thermal and Selleck H 89 tunneling components [48]. The effect of quantum tunneling and electrostatic short channel is not treated, which makes it difficult to study scaling behaviors

and ultimate scaling limits of GNR SB FET where the tunneling effect cannot be ignored [20]. The tunneling current is the main component of the whole current which requires the use of the quantum transport. Close to the source within the band gap, carriers are injected into the channel from the source [49]. In fact, the tunneling current plays a very important role in a Schottky contact device. The proposed model includes tunneling current through the SB at the contact interfaces, appropriately capturing the impact

of arbitrary Rebamipide electrical and physical factors. The behavior of the proposed transistor over the threshold region is obtained by modulating the tunneling current through the SBs at the two ends of the channel [20]. The effect of charges close to the source for a SB FET is more severe because they have a significant effect on the SB and the tunneling possibility. When the KPT-330 cost charge impurity is situated at the center of the channel of a SB FET, the electrons are trapped by the positive charge and the source-drain current is decreased. If the charges are situated close to the drain, the electrons will collect near the drain. In this situation, low charge density near the source decreases the potential barrier at the beginning of the channel, which opens up the energy gap more for the flow of electrons from the source to the channel [50]. Electrons moving from the metal into the semiconductor can be defined by the electron current density J m→s, whereas the electron current density J s→m refers to the movement of electrons from the semiconductor into the metal. What determines the direction of electron flow depends on the subscripts of the current. In other words, the conventional current direction is opposite to the electron flow.

C parapsilosis reference strain ATCC 22019 was used as control

C. parapsilosis reference strain ATCC 22019 was used as control. Statistics Statistical analysis was performed using Instat software (GraphPad, USA). One-way ANOVA followed by Post-hoc test (Bonferroni) was used to selleck evaluate the level of statistical significance of clustering. The association between biofilm and proteinase production was determined by Pearson’s correlation coefficient Proteasome inhibitors in cancer therapy (r). Differences between proteinase/biofilm producers versus non producers were examined using Fisher’s exact test. A P value < 0.05 was considered statistically significant. Results Molecular typing of Candida parapsilosis isolates AFLP was used to confirm correct species identification and to evaluate genetic variability

within the selected 62 C. parapsilosis isolates. AFLP profiles obtained for C. parapsilosis consisted of 80 fragments ranging from 100 to 700 bases. Fragments larger than 700 bases were used as a control of DNA integrity. The number of monomorphic fragments was 62, which were common to all C. parapsilosis isolates. Therefore, these fragments were considered species specific

and used for identification. Indeed, as shown in Figure 1A, which includes a wider panel of clinical isolates, this RG-7388 in vitro method allowed us to identify the presence of C. metapsilosis and C. tropicalis (CP542, CP534, CP557), which were excluded from this study. Identification of C. tropicalis and C. metapsilosis was performed by comparing AFLP profiles with those of 16 different fungal reference species [[16], data not shown]. Figure 1 AFLP patterns. Adenosine triphosphate (A) AFLP profiles obtained from the molecular screening of 48 putative Candida parapsilosis

clinical isolates and reference strains ATCC 22019 (C. parapsilosis), ATCC 96139 (C. orthopsilosis) and ATCC 96143 (C. metapsilosis). In bold, isolates used in this study for genotyping and phenotyping isolated from Argentina (CP540-558) and Hungary (510-536). M 50-500 base molecular weigh standard. In italics, the non-parapsilosis isolates identified during the AFLP screening. (B) AFLP profiles of 34 C. parapsilosis strains isolated from Italy (CP1-CP502) and New Zealand (CP425-486). At the top of the figure, reference strains for C. metapsilosis (ATCC 96143) C. orthopsilosis (ATCC 96139) and C. parapsilosis (ATCC 22019) are included. Figure 1 displays the AFLP profiles obtained from several C. parapsilosis isolates including those selected for the study and isolated from Argentina, Hungary (Figure 1A), Italy, and New Zealand (Figure 1B). When the presence/absence of fragments was the only parameter considered in AFLP analysis, very little genotypic diversity within the isolate collection was found (Figure 1A-B). In fact, the majority of AFLP markers included in the analysis (n = 80) were monomorphic, with only 18 polymorphic fragments. In agreement, UPGMA analysis indicated that all isolates grouped together, with a similarity index (SAB) higher than 0.96 (Figure 2A).

PubMedCrossRef 29 Rogers BA, Sidjabat HE,

PubMedCrossRef 29. Rogers BA, Sidjabat HE, Paterson DL: Escherichia coli O25b-ST131: a pandemic, multiresistant, community-associated strain. J Antimicrob Chemother 2011,66(1):1–14.PubMedCrossRef 30. Karfunkel D, Carmeli Y, Chmelnitsky

I, Kotlovsky T, Navon-Venezia S: The emergence and dissemination of CTX-M-producing Escherichia coli sequence type 131 causing community-onset bacteremia in Israel. Eur J Clin Microbiol Infect Dis 2012,32(4):513–521.PubMedCrossRef 31. Cegelski L, Marshall GR, Eldridge GR, Hultgren SJ: The biology and future prospects of antivirulence therapies. Nat Rev Microbiol 2008,6(1):17–27.PubMedCrossRef Competing interests The authors declared that they have no competing interests. Ro 61-8048 chemical structure Authors’ contributions ID and KP design the study. ID, AK, AÖ and MM-102 mouse BS conducted the experiments. ID, AK, AÖ and KP analyzed the data. ID, AK, BS and KP drafted the article. All authors read and approved the final manuscript.”
“Background Rhizobia are nitrogen-fixing soil bacteria that show intracellular symbiosis with their

host legume. This symbiotic interaction has become a model system to identify and characterize the attractive mechanism employed by invasive bacteria during chronic host interactions [1]. This symbiosis begins with the secretion of flavonoids by the legume. Subsequently, nod genes of rhizobia are activated, and Nod factors (i.e. lipopolysaccharides; LPS) are secreted by rhizobia as signals [2]. After signal exchange between host and symbiont, rhizobia infect the host legume, escaping the vegetative defense responses. The host then produces nodules to maintain symbionts and endocytically incorporates rhizobia into the nodules [3]. In a legume nodule, the host provides C4 dicarboxylates to symbiotic rhizobia as the carbon source; rhizobia fix atmospheric nitrogen and provide ammonia to the host as a nitrogen source in return [4]. Thus, the host plants are able to overcome their nitrogen deficiency. Lotus japonicus and Mesorhizobium loti are model organisms of legume-rhizobia symbiosis. The entire genome structures of L. japonicus MG-20 and M. loti Protein kinase N1 MAFF303099 have been reported

previously [5, 6], and the database is maintained by the Kazusa DNA Research Institute (Rhizobase; http://​genome.​microbedb.​jp/​rhizobase). Transcriptome analysis of M. loti by DNA microarray revealed that most of the transposase genes and nif, fix, fdx, and rpoN on the symbiosis island were highly upregulated under the symbiotic condition, while genes for cell wall synthesis, cell division, DNA replication, and flagella formation were strongly repressed under the symbiotic condition [7]. However, less information is available about M. loti than about other genera of rhizobia, such as Sinorhizobium meliloti, Rhizobium leguminosarum, and Bradyrhizobium japonicum. In addition to transcriptome analysis, proteome analysis has recently attracted much attention.

Oxford University Press, OxfordCrossRef FitzGerald GA (2009) Movi

Oxford University Press, OxfordCrossRef FitzGerald GA (2009) Moving clinical

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